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Selegiline acts as a monoamine oxidase inhibitor, and increases levels of monoamine neurotransmitters in the brain. At typical clinical doses, selegiline is a selective and irreversible inhibitor of monoamine oxidase B MAO-B, increasing levels of dopamine in the brain.
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In humans, brain dopamine is metabolized by MAO-B, and the blockade of this enzyme will reduce the metabolism of dopamine. Patients who received selegiline, with placebo or with tocopherol, experienced a significant delay in the need for levodopa therapy. Patients who received placebo required levodopa at a projected median of 15 months from enrollment, whereas those who received selegiline required levodopa ataprojectedmedianof24monthsafterenrollment.
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